top of page

Inflammatory Myopathies (IIM): Polymyositis and Dermatomyositis “WEAK MUSCLE” Diagnosis, Staging, and Step-Care Treatment Plan By 2017 EULAR/ACR probability score

1 Pathophysiology & Clinical Presentation

Key point

Polymyositis (PM)

Dermatomyositis (DM)

Immune driver

CD8⁺ T-cell–mediated myofibre injury

Humoral + complement attack on endomysial vessels

Core symptom

Symmetric proximal muscle weakness (climbing stairs, hair-combing)

Same muscle weakness plus pathognomonic rashes

Cutaneous clues

Heliotrope eyelid rash, Gottron papules, V-sign, shawl sign, holster sign, CMVE

2 Diagnostic Framework

Mnemonic “W-EMB(S)” – Weakness, Enzymes, Myopathic EMG, Biopsy, (Skin for DM).

Criterion

What to check

A — Muscle weakness

Symmetric, proximal, ≥ 4 weeks

B — Muscle enzymes

CK, AST, ALT, LDH, aldolase ↑

C — EMG

Fibrillation potentials, short polyphasic MUAPs

D — Biopsy

PM → endomysial CD8⁺ & necrosis; DM → perifascicular atrophy

E — Skin

Heliotrope, Gottron, CMVE, V/shawl/holster signs



Diagnosis: PM = A–D present, no E. DM = E + ≥ 3 of A–D. Apply the 2017 EULAR/ACR probability score to label possible/probable/definite  IIM; a score ≥ 7.5 (with biopsy) or ≥ 8.7 (without) = definite myositis.org.

3 Staging & Activity Monitoring

Domain

Tool (+ timing)

Clinically meaningful change

Muscle strength

MMT-8 every visit (0–80)

Response = ≥ 20 % or ≥ 6-point gain sralab.org

Global disease activity

IMACS Core Set Measures → calculate ACR/EULAR Total Improvement Score (TIS)

TIS ≥ 20 = minimal, ≥ 40 = moderate, ≥ 60 = major improvement academic.oup.comniehs.nih.gov

Skin severity (DM)

CDASI activity score

≤ 14 mild, 15–40 moderate, > 40 severe jaad.org

Permanent damage

Myositis Damage Index (MDI) yearly

Higher score = chronic / polycyclic course

Extra-muscular

HRCT + PFTs (ILD), echo ± RHC (PAH), age-appropriate malignancy screen (anti-TIF1-γ, NXP-2)


4 Investigations – “MIOSITIS” checklist

Letter

Order this

Purpose

M

MRI STIR thighs/shoulders

Localise active muscle for biopsy

I

Immune panel (MSA/MAA)

Phenotype & prognosis (e.g., anti-MDA-5 → RP-ILD)

O

Organ screen – HRCT, PFTs, echo, LFTs

Baseline & follow-up

S

Serum enzymes – CK etc.

Disease activity

I

Infection screen – HBV/HCV, HIV, IGRA

Pre-immunosuppression

T

Tumour search > 40 yrs

CT chest/abd/pelvis, mammogram/colonoscopy/PSA

I

Interval labs q 4–8 w on therapy

Response & toxicity

S

Strength testing (MMT-8 / dynamometer)

Objective follow-up


5 Management – Treat-to-Target ≤ 6 months

Step

Drug(s) & key points

Escalate when…

1 High-dose steroid

Prednisone 1 mg/kg/day (max 80 mg) ± IV methyl-pred 1 g ×3 if severe dysphagia/RP-ILD

CK or strength plateaus, unable to taper < 20 mg by 3 mo

2 Steroid-sparing csDMARD (start ≤ 4 wks)

Methotrexate 15–25 mg/wk or Azathioprine 2–3 mg/kg/d. ILD: Mycophenolate 1 g bid / Tacrolimus 1–3 mg bid

Persistent activity or steroid-toxicity

3 IVIG 2 g/kg monthly

Rapid weakness, dysphagia, pregnancy, or refractory DM rash

Re-check after 3 cycles

4 Biologic / targeted

Rituximab 1 g × 2 (RIM trial) myositis.org, Abatacept, JAK-i (Tofacitinib) for anti-MDA-5 ILD, Cyclophosphamide for severe ILD


5 Cutaneous DM adjuncts

Hydroxychloroquine 200–400 mg/d, potent topical steroids, sun-block


6 Rehab & prevention

Early physiotherapy, vitamin D + DEXA, vaccines (flu, pneumococcal, zoster, COVID-19)


Toxicity pearls • MTX → hepatotoxicity & pneumonitis • AZA → TPMT/NUDT15-related cytopenia • Mycophenolate → cytopenia & diarrhoea • Rituximab → HBV reactivation.

6 General Doctor vs Rheumatology – who does what?

Task

GP / Internist

Needs Rheumatologist

Suspicion, baseline CK, ANA, rule-out infection

Start high-dose steroid if profound weakness or ILD

✓ (then phone Rheum same day)

Dose-taper & DMARD plan

Vaccination, osteoporosis prophylaxis, BP/DM care

Routine labs & CK q 4–8 w

✓ (forward to Rheum)

Adjust drugs / interpret flares

Decisions on IVIG, biologics, cyclophosphamide

✓ – complex immunotherapy

Work-up / management of ILD, PAH, malignancy

Arrange tests

Rheum + Respiratory / Oncology

Refractory disease, pregnancy, juvenile cases

Stabilise & refer

Multidisciplinary care

Bottom line: All confirmed or strongly suspected IIM patients require early Rheumatology co-management; the generalist initiates life-saving steroids, screens for comorbidities, and provides long-term preventive care.

7 High-yield Dermatomyositis Rashes

Rash

Clinical clue

Exam mnemonic

Gottron papules

Violaceous papules over MCP/PIP

“Got Guitar Knuckles”

Heliotrope

Lilac eyelid discoloration ± oedema

“Helios = sun-purple”

V-sign / Shawl sign

Photodistributed chest / shoulder erythema

Letter shapes on skin

Holster sign

Lateral thigh erythema

“Gun-holster rash”

CMVE

Confluent macular violaceous erythema

Key board-style patch


One-minute OSCE Script

  1. “Mrs D has symmetric proximal weakness, CK 2400 IU/L, heliotrope rash → 2017 EULAR/ACR score 9.1 = definite dermatomyositis.”

  2. Stage: MMT-8 54/80 (moderate), TIS baseline 0, CDASI 28 (moderate), HRCT clear.

  3. Order MRI thighs, myositis panel, EMG, and malignancy screen.

  4. Start Prednisone 1 mg/kg + Methotrexate 15 mg/wk + calcium/vit-D; refer to Rheumatology within a week; physiotherapy day 1.

  5. Review CK & strength at 4 weeks; add IVIG if TIS < 20.

Use “W-EMB(S)”, TIS 20-40-60, and MIOSITIS check-list to keep both patients and examiners safe.

Diving Deeper into the Core Diagnostic Criteria for Polymyositis (PM) & Dermatomyositis (DM)

Below, each element of the classic A–E framework is unpacked in detail and cross-mapped to the weightings in the 2017 EULAR/ACR probability score (the current research-grade standard).

Criterion

What to document in practice

Key histo-/physiologic pearls

EULAR/ACR weight (with biopsy)

A — Muscle weakness

• Manual Muscle Testing (MMT-8) or dynamometry of neck-flexors, deltoids, biceps, wrist extensors, iliopsoas, gluteus maximus, quadriceps, ankle dorsiflexors.


• Symmetric, proximal, ≥ 4 weeks; neck-flexors weaker than extensors; proximal legs weaker than distal.

CD8⁺ T-cell cytotoxicity in PM; perifascicular ischemia in DM cause predominantly proximal fibre loss.

• Prox UL: 0.7  • Prox LL: 0.5  • Neck-flexor < extensor: 1.6  • Prox > distal leg: 1.2 myositis.org

B — Muscle enzymes

Order CK, aldolase, AST, ALT, LDH at baseline and every taper decision.


Typical CK peaks:


  • PM / DM active: > 1 000 IU (L) (may reach > 10 000).


  • “Amyopathic” DM: CK often normal.

CK tracks necrosis/regeneration; aldolase may rise first as CK normalises. Beware: strenuous exercise, statins and hypothyroidism also raise CK.

CK/LD/AST/ALT↑: 1.4 health.com

C — Electromyography

Needle EMG in ≥ 2 proximal & 1 distal muscle:


Fibrillation/positive sharp waves at rest


Short-duration (< 5 ms), low-amplitude (< 500 µV) polyphasic MUAPs


Early full recruitment with minimal effort

Reflects membrane irritability from inflammatory insult. Complex repetitive discharges & “myotonic-like” potentials bolster diagnosis.

(EMG is not scored in 2017 criteria but remains clinically invaluable) myositis.org

D — Muscle biopsy

Biopsy an MRI-edematous site before steroids if possible. Process fresh-frozen + formalin.

Polymyositis → Endomysial CD8⁺ T-cells & macrophages invading non-necrotic fibres with diffuse MHC-I up-regulation. autoimmunhighlights.biomedcentral.com


Dermatomyositis → Perifascicular atrophy, perivascular/ perimysial CD4⁺ T-cells & B-cells, capillary C5b-9 complement deposition. academic.oup.com

• Endomysial inflam (non-invasive): 1.7


• Perivascular/perimysial inflam: 1.2


Perifascicular atrophy: 1.9 myositis.org

E — Skin manifestations (DM)

Heliotrope rash (eyelids), Gottron papules/sign, V-sign, shawl sign, holster sign, Mechanics hands, periungual telangiectasia. Document photos under good light.

Skin pathology shows interface dermatitis & mucin; MxA and IFN-signature staining aid amyopathic DM Dx.

• Heliotrope: 3.2


• Gottron papule: 2.7


• Gottron sign: 3.7 myositis.org

How the 2017 EULAR/ACR Probability Score Works

  1. Assign points for each variable present (table clip above).

  2. Sum the points.

  3. Convert to a probability of IIM:*Without biopsy: P = 1 / [1 + e^(6.49 – score)]With biopsy: P = 1 / [1 + e^(5.33 – score)] myositis.org

  4. Interpret:

    • ≥ 7.5 (no biopsy ≥ 8.7) → Definite IIM (> 90 % probability)

    • 5.5 – 7.4 (no biopsy 6.7 – 8.6) → Probable IIM (> 55 %)

    • 5.3 – 5.4 → Possible IIM (50–55 %)

    • < 5.3 (< 6.5 with biopsy) → Unlikely

Shortcut rule still acceptable in clinics: PM = A–D + absence of E |  DM = E + ≥ 3 of A–D (Bohan & Peter).

Putting It Together – Sample Calculation

Patient: 46-year-old woman with proximal leg weakness (MMT 4/5), heliotrope rash, neck-flexor 3/5, CK 3500, anti-Jo-1⁺, biopsy shows perifascicular atrophy.

Variable

Points

Age ≥ 40

2.2

Prox-LL weakness

0.5

Neck-flexor weaker

1.6

CK↑

1.4

Heliotrope

3.2

Anti-Jo-1

3.8

Perifascicular atrophy

1.9

Total

14.6

With biopsy:P = 1 / [1 + e^(5.33 – 14.6)] ≈ 0.999 → Definite DM.

Practical Tips for Each Criterion

  • Weakness (A) – always test neck flexors; their relative weakness carries the biggest score (1.6 pts).

  • Enzymes (B) – trend CK every 4–8 weeks; a fall > 80 % yet persistent weakness suggests steroid-induced myopathy rather than active disease.

  • EMG (C) – combine with MRI (STIR) to target biopsy and avoid false-negatives.

  • Biopsy (D) – take from deltoid or vastus lateralis; avoid severely atrophic muscle. Request MHC-I, CD3, C5b-9 immunostains.

  • Skin (E) – photograph lesions; they may fade after steroids, hampering later scoring.

Key Take-aways

  • Weightings matter: heliotrope rash (3.2 pts) or anti-Jo-1 (3.8 pts) can swing a case from “possible” to “definite” quickly.

  • Biopsy deepens certainty and allows subtype classification (PM vs DM vs IBM).

  • Use the web calculator (link in criteria paper) at the bedside for exact probability.

  • Remember the probability concept: the criteria classify for research; clinical judgment still rules when treatment can’t wait for a biopsy.

Feel free to ask for more on biopsy techniques, MRI protocols, or management algorithms!

Recent Posts

See All

Comments

Rated 0 out of 5 stars.
No ratings yet

Add a rating
Post: Blog2_Post

​Message for International and Thai Readers Understanding My Medical Context in Thailand

Message for International and Thai Readers Understanding My Broader Content Beyond Medicine

bottom of page